Researchers at UC San Francisco have uncovered a surprising culprit in brain aging: a protein called FTL1. In aging mice, too much of this protein weakened memory and disrupted neural connections. But when scientists blocked FTL1, the mice’s brains regained youthful function, and their memory tests dramatically improved. Aging Hits the Hippocampus Hard Aging takes […]



Summary

UC San Francisco researchers found excess FTL1 protein contributes to brain aging in mice, specifically weakening memory and disrupting neural connections. Surprisingly, blocking FTL1 reversed these effects, restoring youthful brain function and improving memory test performance in older mice. This discovery suggests that targeting FTL1 could potentially offer a novel therapeutic approach to combat age-related cognitive decline.

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