Nature Immunology, Published online: 29 August 2025; doi:10.1038/s41590-025-02255-y Alatrakchi and colleagues profile immune cells from liver and blood obtained from patients with MASLD/MASH using single-cell sequencing. They note increased immunoregulatory programs that Read More
Tags :dysfunction
Education, Research & Positions
Dysfunction in primate dorsolateral prefrontal area 46 affects motivation and
Science, Volume 389, Issue 6762, Page 840-844, August 2025. Summary Without knowing the specific content of the Science article, a 100-word summary can only speculate based on the general nature of the journal. It’s likely a research Read More
Education, Research & Positions
Multiple Sclerosis May be Related to Mitochondrial Dysfunction – New
Multiple sclerosis (MS) is a chronic inflammatory disorder that is thought to impact about 2.3 million people around the world. .. Summary Multiple sclerosis (MS) is a chronic inflammatory disease affecting approximately 2.3 million people globally. While the Read More
Education, Research & Positions
LARP4-mediated hypertranslation drives T cell dysfunction in tumors – New
LARP4-mediated hypertranslation drives T cell dysfunction in tumors Summary LARP4, an RNA-binding protein, plays a significant role in tumor-induced T cell dysfunction. Researchers discovered that within tumors, T cells exhibit increased LARP4 expression. This Read More
Education, Research & Positions
Glymphatic dysfunction in Alzheimer’s disease: A critical appraisal – New
Science, Volume 389, Issue 6756, July 2025. Summary Without knowing the specific articles or content within that issue of Science (Volume 389, Issue 6756, July 2025), a summary is impossible. A generic summary might say: “The July 2025 issue of Science explores Read More
Education, Research & Positions
CD4<sup>+</sup> T cells license Kupffer cells to reverse CD8<sup>+</sup> T
Nature Immunology, Published online: 30 June 2025; doi:10.1038/s41590-025-02199-3 Here the authors show that CD4+ effector T cells prevent or reverse CD8+ T cell dysfunction by licensing Kupffer cells to trigger IL-27 production, defining Read More
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