Overactivity in the reticular thalamic nucleus was shown to cause autism-like behaviors in mice. Drugs that reduce this activity reversed the symptoms. Researchers at Stanford Medicine studying the neurological basis of autism spectrum disorder have found that excessive activity in a particular brain region may be responsible for driving behaviors commonly linked to the condition. […]



Summary

Stanford Medicine researchers found that overactivity in the reticular thalamic nucleus (RTN) in mice caused autism-like behaviors. This discovery suggests a potential neurological basis for autism spectrum disorder. The study showed that reducing RTN activity with drugs effectively reversed these behaviors in the mice. This points to the RTN as a possible therapeutic target for treating autism, offering a potential avenue for developing new medications and interventions. The research provides valuable insight into the complex neurological underpinnings of autism and may pave the way for more targeted treatments.

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