Scientists identified a nuclear mechanism that distinguishes chronic inflammation from acute inflammation in mice.



Summary

Researchers have uncovered a key nuclear mechanism differentiating chronic and acute inflammation in mice. By studying changes within cell nuclei, they identified specific molecular events that drive the transition from a short-term, beneficial immune response (acute inflammation) to a persistent, damaging state (chronic inflammation). This discovery offers a potential target for developing new therapies that can specifically interrupt the progression to chronic inflammatory diseases by manipulating nuclear processes and preventing the switch from acute to chronic stages. This research focuses on the understanding of the molecular basis of inflammation.

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